NDA
Bejelentkezés
Kapcsolat
Differential Induction of Phenylpropanoid Metabolites in Suspension-Cultured Cells of Sugarcane by Fungal Elicitors |
Tartalom: | http://real.mtak.hu/64645/ |
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Archívum: | MTA Könyvtár |
Gyűjtemény: |
Status = Published
Type = Article |
Cím: |
Differential Induction of Phenylpropanoid Metabolites in Suspension-Cultured Cells of Sugarcane by Fungal Elicitors
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Létrehozó: |
Ramesh Sundar, A.
Vidhyasekaran, P.
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Kiadó: |
Akadémiai Kiadó
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Dátum: |
2003
info:eu-repo/date/embargoEnd/2023-04-30
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Téma: |
QH540 Ecology / ökológia
QK10 Plant physiology / növényélettan
QL01 Systematic zoology / állatrendszertan
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Tartalmi leírás: |
The effect of elicitors isolated from the mycelial walls of Colletotrichum falcatum (the red rot pathogen of sugarcane) and from C. lindemuthianum (a non-pathogen) in suspension-cultured cells of sugarcane was studied. Both the elicitors induced the synthesis of enzymes of the phenylÂpropanoid pathway such as PAL, TAL and 4CL and also resulted in the enhanced accumulation of phenolics. However, a specific induction of the defense parameters at higher levels was recorded in suspension cells treated with the pathogen elicitor and no such differential response was observed in the case of the non-pathogen elicitor. Elicitor induced necrosis and browning of cells were observed which suggests an additional evidence that elicitors simulate pathogen infection and thus provide a valuable reason that study on elicitor induced responses may be useful in understanding the host defense mechanisms against the red rot pathogen at molecular level.
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Nyelv: |
magyar
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Típus: |
Article
PeerReviewed
info:eu-repo/semantics/article
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Formátum: |
text
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Azonosító: |
Ramesh Sundar, A. and Vidhyasekaran, P. (2003) Differential Induction of Phenylpropanoid Metabolites in Suspension-Cultured Cells of Sugarcane by Fungal Elicitors. Acta Phytopathologica et Entomologica Hungarica, 38 (1-2). pp. 29-42. ISSN 0238-1249
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Kapcsolat: |
https://doi.org/10.1556/APhyt.38.2003.1-2.5
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Létrehozó: |
info:eu-repo/semantics/embargoedAccess
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